Sex and the Female Brain: Protein in Semen Acts On Female Brain to Prompt Ovulation
ScienceDaily (Aug. 20, 2012)
Excerpt: “Using a variety of techniques, the researchers compared OIF and NGF and found them to have the same size and to cause the same effects across species.”
The same effects across species literally screams CORRELATE, not cause and effect. It seems very unlikely that transport of OIF would occur and lead to the epigenetic effects on the GnRH neuronal system that prompt the LH increase, which is typically associated with the epigenetic effects of mammalian pheromones. The LH increase is also neuroendocrinologically linked to sex differences in estrogens and testosterone that are important to classically conditioned effects on other hormones (e.g., oxytocin and dompamine) and their affect on properly timed mammalian reproductive sexual behavior. Thus, the question is “Do these people really think its a protein in semen that acts on the female brain? If so, is there a model for that — in any species with or without a brain? How did they eliminate the possibility that OIF is a protein that is dependent on the ability of pheromones to classically condition whatever effect it has on ovulation?
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.