Weaponized Health Communication (2)

By: James V. Kohl | Published on: July 17, 2019

Metabolic perturbations and cellular stress underpin susceptibility to symptomatic live-attenuated yellow fever infection  7/15/19

They link an attenuated virus in a yellow fever vaccine from induced effects on the endoplasmic reticulum and the innate immune response to the pathology common to other flaviviral illnesses via Nazi-like experiments on people in Cambodia.

The Nazis, however, did not understand the claims that Schoedinger made famous in “What is Life?” (1944).

In 2018, Nobel Laureate, Ben Feringa put those claims into the context of prophesy from the Holy Bible.

See: Bernard Feringa: The Future of Chemistry – Schrödinger at 75: The Future of Biology

Disabled were walking again, the blind were seeing again, and the death rose from the grave. 2013 was the year in which prophesy from the bible became reality. — De Correspondent dec 2013

See for comparison: Increased adaptive immune responses and proper feedback regulation protect against clinical dengue 8/30/17

Although dengue hemorrhagic fever can be life-threatening, not all dengue virus infections even present with symptoms. To determine what may be driving the differences in clinical and asymptomatic infections, Simon-Lorière et al. examined the serum and immune gene transcripts of Cambodian children infected with dengue virus serotype 1. They saw that relative to those with clinical infections, the small subset of asymptomatic children had increased signs of antigen presentation, T cell activation, and T cell apoptosis; plasmablast differentiation and anti-dengue antibodies seemed relatively lower. These results provide clues for pathways that may drive pathologic responses in severe dengue virus infections.

The link from food energy-dependent pheromone-controlled feedback loops  to the transgenerational epigenetic inheritance of the adaptive immune system could not be clearer than when it was reported as: Feedback loops link odor and pheromone signaling with reproduction 11/18/05

Attempts to stop the Nazi-like experiments were reported in the fake news as: Exclusive: Trump to order drive for improved flu vaccine

Trump’s early 2018 conversation with Bill Gates on the need for a universal flu vaccine also shifted the president’s thinking, said three individuals with knowledge of the strategy.

Who are the three individuals touting this nonsense? Are they the paid idiot minions of someone like Bill Gates with conflicts of interest that extend globally?

Bill Gates: Trump ‘super interested’ in universal flu vaccine (4/30/18)

Gates…told him [President Donald Trump] that he should associate himself with “American innovation.” The interview comes just days after the billionaire philanthropist pledged $12 million in funding for the vaccine.

Oh my God. Does Bill Gates wants President Donald Trump to do what the Nazis did? Experiment with vaccines?

Top 10 Horrific Nazi Human Experiments 6/26/17
Spotted Fever Experiments
From late 1941 to early 1945, physicians at Buchenwald and Natzweiler concentration camps conducted experiments on inmates for the benefit of the German armed forces. They were testing the effectiveness of spotted fever and other vaccines.

Approximately 75 percent of the subjects were given trial vaccines for spotted fever or nourished with other chemical substances. They were then injected with the virus. As a result, more than 90 percent of them died.[9]

The virus was introduced to the remaining 25 percent without any prior protection as a control group. Most of them didn’t make it. The physicians also conducted experiments associated with yellow fever, smallpox, typhus, and other diseases. Hundreds of inmates died, and many more suffered unbearable pain as a result.

Malaria Experiments
For over three years, a series of experiments was conducted on more than 1,000 inmates at Dachau concentration camp in search of a treatment for malaria. Healthy inmates were infected by mosquitoes or extracts from those mosquitoes.

Subjects who contacted malaria were then treated with various drugs to test their efficacy. Many inmates died. Those who lived suffered painfully, and most were permanently disabled.[12]

Is Bill Gates promoting the Nazi-like testing of vaccines on children in Cambodia?  That’s where the experiments were performed in Simon-Lorière et al., who examined the serum and immune gene transcripts of Cambodian children infected with dengue virus serotype.

For comparison, President Donald Trump and researchers from Iran are asking questions such as this: Why have microRNA biomarkers not been translated from bench to clinic? 1/17/19

See also: PTEX component HSP101 mediates export of diverse malaria effectors into host erythrocytes 7/16/14

Reported as: Scientists find way to trap, kill malaria parasite

…in the same issue of Nature, also highlights the importance of the pore to the parasite’s survival. Researchers believe blocking the pore leaves the parasite fatally imprisoned, unable to steal resources from the red blood cell or dispose of its wastes.

The issue became one of drug development instead of vaccines. Apparently, there is no need for experiments with vaccines like those that the Nazis performed if money can be made via drug development.
But see also Scientists uncover mechanism behind development of viral infections 7/16/19

These findings also imply that preventing conditions known to place cells under increased stress and altered metabolism, such as obesity, could prevent disease from viral infections.

If vaccines are not required to control the viral infections linked to stress-altered metabolism and obesity, the development of viral infections must somehow be biophysically constrained.

For comparison, the energy-dependent reassembly of viruses prevents development of effective vaccines.That means that neither vaccines or drug development will effectively prevent the pathology caused by viruses.

Viruses Can Scatter Their Genes Among Cells and Reassemble 5/21/19

That is why President Donald Trump is forcing others to look at the data before the focus changes to proper nutrition in the context of two published works that clearly link the creation of amino acids from RNA interference (RNAi) to biophysically constrained viral latency and healthy longevity:

See: Get Well in the RNAi Way-RNAi, A Billion Dollar Baby in Therapy 3/3/16

RNAi has targeted exogenous genes in models of viral infection…”

See also the patent application for biophysically constrained viral latency: RNA-Guided Human Genome Engineering 

5. Repetitive elements or endogenous viral elements can be targeted with engineered Cas+gRNA systems in microbes, plants, animals, or human cells to reduce deleterious transposition or to aid in sequencing or other analytic genomic/transcriptomic/proteomic/diagnostic tools (in which nearly identical copies can be problematic).

Substitutions Near the Receptor Binding Site Determine Major Antigenic Change During Influenza Virus Evolution 11/22/13

Koel et al. (p. 976) show that major antigenic change can be caused by single amino acid substitutions.

Identification of Amino Acid Substitutions Supporting Antigenic Change of Influenza A(H1N1)pdm09 Viruses 1/21/15

This study identified substitutions in A(H1N1)pdm09 viruses that support escape from population immunity but also suggested that the number of potential escape variants is limited by previous exposure to seasonal A(H1N1) viruses.

Simply put, they identified substitutions that link the transgenerational epigenetic inheritance of biophysically constrained viral latency to healthy longevity in all mammals via microRNAs in milk exosomes.

Indeed, Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems (4/11/18) presciently summarized what is currently known to all serious scientists about biophysically constrained viral latency. The EDAR V370A allele is the link from microRNAs in milk exosomes to viral latency in the mouse to human model of how RNA-mediated cell type differentiation protects the supercoiled DNA of all organized genomes from viruses.


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