Vitamin A’s Influence on Immunity
Exposure to vitamin A in the womb influences immune system development and lifelong ability to fight infections, a mouse study shows.
By Ashley P. Taylor | March 19, 2014
Excerpt: “The present study is the first to suggest that this development can be altered by maternal behavior.”
My comment: There’s a model for that! It links nutrient stress and social stress to mammalian immune system function and nutrient-dependent pheromone-controlled ecological adaptations via conserved molecular mechanisms in species from microbes to man.
Nutrient-dependent/pheromone-controlled adaptive evolution: a model
Excerpt: “Embryos with retinoic acid receptor mutations had a smaller percentage of inducer cells than controls.”
My comment: That suggests the mutations do not contribute to adaptive evolution. Does anyone who is not a population geneticist think that mutation-driven evolution is biologically plausible?
Like all other experimental evidence, I think the results from this study show that ecological variation enables ecological adaptations. For contrast, only evidence from population genetics suggests that mutation-initiated natural selection is possible — and that evidence is not experimental evidence. Therefore, the idea that vitamin A causes mutations may seem like pseudoscientific nonsense to some molecular biologists, but not to those who believe in mutation-driven evolution.
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.