Excerpt: The misrepresentations in this video will contribute to the suicides of ~22 veterans each day.
The misrepresentations in this video will contribute to the suicides of ~22 veterans each day.
See for comparison: Genome-wide association study of depression phenotypes in UK Biobank identifies variants in excitatory synaptic pathways
This was reported as Genes that May Trigger Depression Identified
All three reports, from the least technical video representation to the most technical journal article, fail to link the creation of sunlight from microRNA biogenesis to fixation of amino acid substitutions.
For comparison, all serious scientists know that quantized energy-dependent fixation of amino acids stabilizes the organized genomes of all cell types in all living genera. The amino acid substitutions protect people from the virus-driven degradation of messenger RNA, which links mutations to all pathology.
Psudoscientists and other theorists cannot link the claim that Slc6a15 amplifies or reduces stress to the claim that it is among the 5 gene variants linked to suicide.
All serious scientists could search for Slc6a15 and reveal that scientific creationists in South Korea have linked the amino acid transporter transporter gene Slc6a15, to the integrity of brain structures and prevention of depression.
See also: HHV-6 encoded small non-coding RNAs define an intermediate and early stage in viral reactivation 9/5/18
Reported as: Viruses Under the Microscope 9/14/18
“We have identified several viral microRNA molecules which are produced both during active infection and viral activation,” Prusty explains. MicroRNAs directly influence cell metabolism. The RNA assures the flow of genetic information from nuclear DNA into the cell where it is “translated” to proteins. The microRNAs have a regulatory function in this process. They can dock to RNA molecules and prevent them from being translated to proteins or initiate the degradation of RNA molecules. Prusty is certain that the detection of these viral microRNAs can serve as an ideal biomarker under clinical conditions.
That claim can be placed into the context of my life’s works on food energy-dependent pheromone-controlled viral latency in species from microbes to humans via the claims in the cell biology game “Cytosis” for ages 10+.
See also, from the Neuroscience FB group:
Jan Viljoen:
If the genes are so bad, why did evolution not weed it out?
See also from the Journals and Conferences on Genetics Molecular-Biology FB page
FGF2 Has Distinct Molecular Functions from GDNF in the Mouse Germline Niche
The quantized energy-dependent creation of microRNAs links microRNA biogenesis to biophysically constrained viral latency and the creation of FGF2, which “…contributes to induction of differentiation rather than maintenance of undifferentiated spermatogonia, indicating reconsideration of the role of FGF2 in the germline niche.”
Minimal reconsideration of top-down causation links the creation of the sun’s anti-entropic virucidal energy “From Fertilization to Adult Sexual Behavior” via the details in our 1996 Hormones and Behavior review of RNA-mediated cell type differentiation. See: http://www.hawaii.edu/PCSS/biblio/articles/1961to1999/1996-from-fertilization.html
Significance: The creation of anti-entropic virucidal light is linked to all biophysically constrained biodiversity on Earth via the physiology of reproduction in species from yeasts to mammals via the amino acid substitutions that stabilize the cell types of all cells in all individuals of all species.
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.