Genes Aren’t Just Architects; They’re Actors by David Dobbs •
Excerpt: “Robinson and others have for years been waving their hands, pointing at results like this, and arguing that the endgame in genetics is not in differences in what genes we carry, but — especially when most of our genes our shared — in differences in how and when the shared genes express themselves. This is important in understanding differences within a species, whether it be honey bees or humans.”
My comment: In 2000 Elekonich and Robinson extended our 1996 mammalian model of molecular epigenetics and hormone-organized / hormone-activated behavior to insects, with the honeybee as the model organism. I’ve since linked human pheromones to the conserved molecular mechanisms of species from microbes to man in two published papers:
Nutrient-dependent/
I’m not sure how anyone else could explore the topic of species differences in more detail than I have provided during that past two decades since my first presentation to a scientific forum (in 1992) and book publication in 1995. I am sure that the differences in the brain and social behavior of insects and humans are nutrient-dependent and pheromone-controlled. There is no other model for that! The fact that the model is fully detailed in the journal ‘Socioaffective Neuroscience & Psychology’ may be the best indicator that David Dobbs is on the same track as I have been since I first began researching human sexuality in the early 1980s.
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.