The tipping point (revisited): 87K (2)

Fakes expose themselves on Twitter

 Koel et al. (p. 976) show that major antigenic change can be caused by single amino acid substitutions.

The antigenic changes have been linked to Emergence of Human G2P[4] Rotaviruses in the Post-vaccination Era in South Korea: Footprints of Multiple Interspecies Re-assortment Events as portrayed in the 2011 medical thriller “Contagion.”

Vaccine-related morbidity and mortality was transferred to diseases associated with cell type differentiation in other cell types of other tissues across kingdoms. Theorists seem to know nothing about this, and they’re not going to discuss it.

See: A multicellular way of life for a multipartite virus (3/12/19)

Our observation deviates from the classical conceptual framework in virology and opens an alternative possibility (at least for nanoviruses) where the infection can operate at a level above the individual cell level, defining a viral multicellular way of life.

Reported as: Viruses Can Scatter Their Genes Among Cells and Reassemble (5/21/19)

Zwart expects that new theoretical models will come out soon to explore these insights, such as ways of framing the evolution of these viruses in terms of multiple levels of natural selection. Within an individual host plant, local forces of natural selection will allow the virus to balance the production rates of its segments successfully. But when the virus moves on to a new plant, it must be able to adapt to that new host environment as well, so it needs to retain some versatility. A higher level of selection may therefore sometimes temper the local level and rebalance the ratio of segments more evenly.

The claim about new theoretical models that explore the evolution of viruses is an attempt to obfuscate the facts about biophysically constrained viral latency, which prevents the hecatombic evolution of all pathology.

See:  Virus-mediated archaeal hecatomb in the deep seafloor and  Eukaryotic plankton diversity in the sunlit ocean.

All biodiversity is energy-dependent and the energy comes from sunlight. Theoretical models avoid the claim about sunlight as the source of all energy-dependent life on Earth. The models exemplify human idiocy, and all serious scientists know that.

See:

I’ve linked my claims to Feynman’s claims about energy in Nutrient-dependent Pheromone-Controlled Ecological Adaptations: From Angstroms to Ecosystems  (4/11/18)

To support my claims about how viruses in vaccines and viruses used in gene-editing cause morbidity and mortality, I cited published works on brain cancer and Alzheimer’s from “Nature” in this Tweet: Vaccines cause the virus-driven degradation of messenger RNA linked from the creation of toxic peptides to mutations and pathology. That should be stopped before you or someone you love gets brain cancer or Alzheimer’s.

  1. Stem cell-associated heterogeneity in Glioblastoma results from intrinsic tumor plasticity shaped by the microenvironment (4/16/19)
  2. The viral protein corona directs viral pathogenesis and amyloid aggregation (5/27/19)

Biologically uninformed science idiots will not discuss the facts that link the virus-driven creation of toxic peptides from vaccines to pathology because they profit from disease. That’s also why the examples of human idiocy will not discuss that fact that gene-editing uses viruses that cause morbidity and mortality across kingdoms.

But, see these reports from 5/27/19.

  1. Cancer cells are quick-change artists adapting to their environment and
  2. Viruses wearing ‘protein coats’ are more infectious, linked to Alzheimer’s disease

The virus-driven degradation of messenger RNA that causes antigenic changes in all cell types has been linked from changes in cancer cells to all neurodegenerative diseases, not just Alzheimer’s.

See also: A Programmed Dual-Functional DNA Tweezer for Simultaneous and Recognizable Fluorescence Detection of microRNA and Protein (5/28/19)

The significant variation of fluorescence intensity enables one-step, cost-effective and specific quantization of miRNA 21 and MUC1 with high sensitivity down to 32 fM and 2.6 fg.mL-1 (8.5 pM), respectively. The novel DFDT-based assay route of multiplex analytes is promising and has the potential for rapid and reliable diagnosis and treatment of cancer-related diseases.

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Author: James Kohl

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