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These novel studies form an interesting backdrop for the paper by Maiese that discusses exciting prospects for clinical treatments with non-coding RNAs and their association with circadian clock genes, forkhead transcription factors, SIRT1, and mTOR. Each of these pathways have a critical relationship with the programmed death pathways of autophagy and apoptosis that affect neuronal stem cell development and neuronal differentiation, cognitive function, and vascular disease.
The treatments with non-coding RNAs link the creation of quantized energy to microRNA biogenesis. MicroRNAs biophysically constrain viral latency in the context of everything known to serious scientists about nutritional epigenetics and healthy longevity. Only pseudoscientists and other biologically uninformed theorists have failed to link levels of quantized energy-dependent biological organization to disease prevention.
Biologically uninformed science idiots, who failed to grasp the importance of the levels of biological organization, must now change their approach to treating all diseases.
Quantized energy stabilizes microtubules. That fact has been linked to the treatment of Alzheimer’s in the context of everything known to all serious scientists about biophysically constrained viral latency.
See also: Anesthesia Points to Deeper Level ‘Quantum Channels’ as Origins of Consciousness Subtitle: Electron resonance pervades the brain
Using molecular modeling, the US-Canadian team of Travis Craddock, Stuart Hameroff and Jack Tuszynski had previously shown anesthetic binding in ‘quantum channels’, non-polar arrays of amino acid pi resonance clouds, winding through microtubules.
From the concluding paragraph:
Future work on O. vulgaris olfaction must also consider how animals acquire the odours detected by the olfactory organ and what kind of odour the olfactory organ perceives. The OL acting as control centre may be target organ for metabolic hormones such as leptin like and insulin like peptides, and olfactory organ could exert regulatory action on the OL via epigenetic effects of nutrients and pheromones on gene expression (Kohl, 2013; Elekonich and Robinson, 2000). — p. 61
Kohl (2013) was my review. Elekonich and Robinson (2000) cited our 1996 review of RNA-mediated cell type differentiation and extended what was know from all invertebrates to all vertebrates. We had already linked yeasts to mammals and Turin’s group linked changes in electron spin to biophysically constrained ecosystem via effects of anethesia in Drosophilia.
It is time to ask yourself and others why this information appears to be relatively unknown. Sometimes it appears likely that a conspiracy of ignorance prevents dissemination of facts. No other conspiracy is required.
Reduced expression of brain-enriched microRNAs in glioblastomas permits targeted regulation of a cell death gene Has your death gene been activated, yet?
Is your brain still fully functional?
Mitochondrial genetics has several unique aspects to its analysis, and corresponding special considerations. Here, we review the components of mitochondrial genetic analysis – nuclear DNA, mitochon-drial DNA, mitochondrial pathways, pseudogenes, nuclear-mitochondrial mismatch, and microRNAs – that could contribute to an observable clinical phenotype.
Eat something or die: Plant MicroRNAs in Cross-Kingdom Regulation of Gene Expression
Protect yourself from the virus-driven degradation of messenger RNA that serious scientist have linked from mutations to all pathology.