Social isolation disrupts myelin production.
Instead of simply offering more evolutionary theory, we detailed the link from the sensory environment to sexual orientation (e.g., the epigenetic effects of pheromones on the gonadotropin releasing hormone neuronal system).
Ingestive behavior and social behavior are odor receptor-mediated in all species (not just those that are sensitive to light) as would be expected due to the common molecular biology of all species.
Kudos to them for moving us forward and away from random mutations theory to an era where geneticists and neuroscientists can examine sensory cause and effect in the proper perspective of an epigenetic continuum of unconscious affects on genomic interactions…
Do you think if I told others that pheromones are like sugar (instead of spices) they would develop a taste for learning about what’s required to link sensory input – like food odors and pheromones- directly to hormones like GnRH and animal behavior?
This is but one of many studies that link the epigenetic effects of nutrient chemicals and pheromones to adaptive evolution via classically conditioned behaviors directly linked from olfactory/pheromonal input to brain development and plasticity altered by stress.
Taken together, these two open-access articles from the supplement offer a concise and reasonably accurate overview of the receptor-mediated events that are required for adaptive evolution.
The idea that ecological niches and social niches are the determinants of neurogenic niches, like those that develop with exposure to food odors and social odors, is one that is important to consider whether we intend to look at pharmacogenomics or to better understand the development of human behavior in the context of epigenetic effects of odors on brain development.
1) ecology and the nutrient chemical-dependent evolution of ecological niches;
2) social interactions and the pheromone-dependent evolution of social niches.