a model for adaptive evolution, for example, that is not nutrient chemical-dependent and pheromone-controlled?
1996 “It is now recognized that other genes on other chromosomes can induce sex reversal regardless of the individual’s SRY status.
2012 “…we now know that the SRY can begin directing sexual differentiation prior to onset of hormonal secretions
Social isolation disrupts myelin production.
the bottom-up epigenetic effects of nutrient chemicals on speciation that are concurrently controlled by the top-down epigenetic effects of pheromones on reproduction challenges those who might rather attempt to-explain away the conserved molecular biology of adaptive evolution via random mutations theory
the logic of gene regulatory networks in adaptive evolution does not skip any species.
Stochastic gene expression is not random; it results from nutrient chemicals like glucose entering the cell.
We incorporated what is currently known about the ability of chemical signals to condition behavior. This conditioning occurs via epigenetic effects that calibrate and standardize the molecular biology of intracellular signal transduction and stochastic gene expression