the honeybee model organism links the epigenetic effects of nutrient chemical-dependent pheromone-controlled adaptive evolution to genes, behavior, and back in species from microbes to man.
Public release date: 11-Dec-2012 National Institute for Mathematical and Biological Synthesis ) Study finds epigenetics, not genetics, underlies homosexuality Excerpt: “…researchers from the Working Group…
the bottom-up epigenetic effects of nutrient chemicals on speciation that are concurrently controlled by the top-down epigenetic effects of pheromones on reproduction challenges those who might rather attempt to-explain away the conserved molecular biology of adaptive evolution via random mutations theory
I mention this only to note the likelihood that random mutations are not driving the disease processes — like Alzheimer’s and that proper nutrition and pheromone therapy offers hope to an aging population.
By the time you get to 4 or 5 million nutrient-chemical and pheromone-controlled DNA switches in the human genome
A detailed model exemplifies the effects of olfactory/pheromonal conditioning, which alters genetically predisposed, nutrient chemical-dependent, hormone-driven mammalian behavior and choices for pheromones that control reproduction via their effects on luteinizing hormone.
Does anyone think that what’s reported here exemplifies an effect of a random mutation on ‘junk DNA’?
epistasis is achieved via the epigenetic effects of nutrient chemicals, which are balanced by the epigenetic effects of their metabolism to pheromones that control reproduction in species from microbes to man.
The epigenetic effects of nutrient chemicals on gene duplication and species diversity and the ability to metabolize nutrient chemicals is controlled by the metabolism of the nutrient chemicals to pheromones that epigenetically control reproduction in all species