A detailed model exemplifies the effects of olfactory/pheromonal conditioning, which alters genetically predisposed, nutrient chemical-dependent, hormone-driven mammalian behavior and choices for pheromones that control reproduction via their effects on luteinizing hormone.
It is no fun to be ignored, but that seems to be a common problem for those who challenge theories, whether or not those theories make sense in the context of biological facts.
We incorporated what is currently known about the ability of chemical signals to condition behavior. This conditioning occurs via epigenetic effects that calibrate and standardize the molecular biology of intracellular signal transduction and stochastic gene expression
Earlier this year I addressed the commonalities and the adaptive evolution of differences in systems-level and behavioral responses in the context of the FDA Critical Path Initiative and ASAM policy statement.
The link to appetite regulation is from olfactory (and probably pheromonal) stimuli directly to the diet-responsive (and probably pheromone-responsive) hypothalamic neurogenic niche that controls nutrient chemical acquisition via its control of the dopaminergic and serotoninergic neuronal systems
Their results exemplify how olfactory/pheromonal conditioning of responses paired with with other sensory input occurs.