Schizophrenia’s Intricacies
By Abby Olena
Two studies provide insight into the genetics of the disorder and show again how complex it is.
Excerpt: “Even small new mutations were not randomly occurring, but were concentrated in a relatively small number of genes that are crucial to the way nerve cells communicate across synapses.”
My comment: Predictably, someone will soon link these non-randomly occurring new mutations to the nutrient-dependent pheromone-controlled sans the theory of mutation-driven evolution. For example, in the article Mosaic Copy Number Variation in Human Neurons, we read “One straightforward hypothesis is that neurons with different genomes will have distinct molecular phenotypes because of altered transcriptional or epigenetic landscapes.”
See also:
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.