“Fluctuating environment may have driven human evolution.” December 24th, 2012.
Excerpt: “Changes in food availability, food type, or the way you get food can trigger evolutionary mechanisms to deal with those changes. The result can be increased brain size and cognition, changes in locomotion and even social changes—how you interact with others in a group. Our data are consistent with these hypotheses.”
My comment: I think what he meant to say was that their data are consistent with my model, which details cause and effect at the molecular level of adaptive evolution. That’s where nutrient chemicals and their metabolism to pheromones epigenetically controls adaptive evolution via nutrient chemical-dependent pheromone-controlled reproduction in species from microbes to man.
See for example: Kohl, J.V. (2012) Human pheromones and food odors: epigenetic influences on the socioaffective nature of evolved behaviors. Socioaffective Neuroscience & Psychology, 2: 17338.
Or the diagram of the model: Human Pheromones: Epigenetic Effects of Odors and Their Affects on Behavior
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.