When I encounter a relatively new term such as mitochondrial gene fragment that might be used instead of the term microRNA, I check the PubMed data base for attempts at obfuscation.
For example, see: mitochondrial gene fragments+ microRNA Items: 6
If the mitochondrial gene fragments are microRNA flanking sequences, more than 81,000 publications may link them to biophysically constrained viral latency in species from microbes to humans via the creation of the sun’s anti-entropic virucidal energy. For example, the electron transport chain links the creation of subatomic particles to all biodiversity via light-activated microRNA biogenesis in plants, naturally occurring RNA interference, and the physiology of reproduction.
The pathway from changes in angstroms to ecological adaptations in ecosystems has been detailed.
See: Meeting abstract: Population genetics of the electron transport chain in snake populations exhibiting divergent resting metabolic rates (1/5/19)
The electron transport chain (ETC), the centre for ATP production, is composed of 13 mtDNA-coded proteins and 73 nDNA-coded proteins.
The differences in the energy of two photons and the differences in the energy of electrons were linked to the biophysically constrained energy-dependent creation of these functional proteins via changes in base pairs (single nucleotide polymorphisms) and changes in ND5 and CYTB, which were linked from changes in two amino acids to highly segregated differences in two different types of snake populations. Are ND5 and CYTB mitochondrial gene fragments or are they microRNA flanking sequences?
In either case:
“This striking [light-activated difference in] population structure at the mtDNA is in contrast to the low genetic structure seen in the background nDNA.
That claim clearly links differences in the biophysically constrained energy of two photons to the differences in base pairs and differences in amino acid substitutions, which were linked to differences in snake populations via comparisons of mtDNA-coded and nDNA-coded ETC genes.
We now incorporate analyses from the nDNA-coded ETC genes, comparing several bioinformatics approaches for sequence assembly and extraction of SNP data. Contrasts between the variation in mtDNA-coded and nDNA-coded ETC genes are made at the level of the gene, protein, and ETC complex and interpreted in the context of mitochondrial physiology and function.
The electron transport complex (ETC) genes link the energy-dependent creation of subatomic particles in sunlight from the creation of ATP-linked variation in mtDNA-coded and nDNA-coded ETC genes to the creation of RNA, enzymes, receptors and behavior at every level of biological organization that links energy-dependent RNA-mediated fixation of amino acid substitutions to species-specific morphology and behavior via sympatric speciation in species from microbes to humans.
My summary: Finally, the energy-dependent creation of the ETC complex can be linked from the creation of subatomic particles to mitochondrial physiology and function without claiming that any functional structure of a protein automagically evolved after energy somehow emerged from the void of nothingness.
This idiotic claim can be eliminated:
Amino acid composition of proteins varies substantially between taxa and, thus, can evolve.
— From Jordan et al., (2005) A universal trend of amino acid gain and loss in protein evolution
We used three mitochondrial gene fragments (COI, Cytb, and ND5) and one nuclear gene fragment (EF-1α) to clarify the invasion history of A. dohrni and assess the effects of geographic events and associated ecological adaptation on the distribution pattern.
The meeting abstract and publication on insect mitochondrial gene fragments Cytb, and ND5, which link the creation of light to the electron transport chain and ecological adaptations, are not attempts to start by eliminating consideration for the creation of subatomic particles.
Instead, the pathway may eliminate claims linked to population genetics such as the claim that amino acids evolve. The current information on the electron transport genes is consistent with what can be learned about biophysically constrained healthy longevity by ages 10+ via games: Subatomic and Cytosis
Ages 10+ can learn how energy-dependent changes in angstroms to ecosystems are linked to ecotypes in the assassin bug and in snakes. Then, ages 10+ can link ecological adaptations to the ecotypes and prevention of the virus-driven degradation of messenger RNA, which serious scientists have linked from mutations to all pathology.
My poster is up! Feel free to check it out at P-29 and swing by during the poster session this afternoon to chat about #PopulationGenetics of #MitoNuclear loci between two remarkable snake eco types at Eagle Lake, CA. #SICB2019 pic.twitter.com/O8kEetIquA
— Randy Klabacka (@HumbleHerper) January 5, 2019
I wrote: This is the best I’ve seen come from #SICB2019. I’d like to use it as a picture perfect example of the fact that others have failed to link the creation of subatomic particles to the ecotypes via microRNA-mediated amino acid changes and sympatric speciation via “…sequence assembly and extraction of SNP data. Contrasts between the variation in mtDNA-coded and nDNA-coded ETC genes are made at the level of the gene, protein, and ETC complex and interpreted in the context of [light-activated] mitochondrial physiology and function.”
@HumbleHerper I would like your permission and the permission of your co-presenters to display your #SICB2019 poster as a picture with the abstract information on my domain https://t.co/E1zDQEt4WY in the context of the series on light and life at base pair resolution. pic.twitter.com/PPfhgBIn2N
— James V. Kohl (@microRNApro) January 5, 2019
@HumbleHerper I would like your permission and the permission of your co-presenters to display your #SICB2019 poster as a picture with the abstract information on my domain https://microrna.pro/ in the context of the series on light and life at base pair resolution.
See for comparison: What If Bacteria Became Antibiotic Resistant?
I wrote: Bacteria adapt to the effects of viruses on the degradation of messenger RNA, which causes all diseases. Continuing to claim that bacteria “evolve” is the path taken by biologically uninformed science idiots since de Vries defined the term “mutation” at the turn of the 20th century. Here we are, more than 100 years later, and you are watching what happens when so-called science journalism confuses ridiculous theories with facts about antibiotic resistance.
Melinda Heal claimed Hashem Al-Ghaili is a Biotechnology Scientist with his Masters. Are you calling him a ‘science journalist’?
She criticized me for what she portrayed as my beliefs in the context of ridiculous assumptions:
You are speaking as though what you state is 100% fact for every single thing. Being a good scientist also entails not discounting that which is unknown.
Do we know for a fact the origin of life was the deep sea vents with the volcanic heat and spewing out the right ingredients? No, but it’s the best theory we have.
Do we know for a fact that Archaea was was the first ever cell? No, but it’s most likely.
Do we know for a fact that eukaryotic cells evolved from prokaryotic cells?
Etc etc etc
I think you are making some very bold claims in your comments, blanket claims, and it just isn’t so. It’s YOUR beliefs.
I replied: Thanks. Yes. He’s a science journalist. For comparison, my publication history and history of presentations since 1992 have established the difference between science journalists and those who have linked the creation of the sun’s anti-entropic virucidal energy to all biophysically constrained biodiversity on earth. See for example: Population genetics of the electron transport chain in snake populations exhibiting divergent resting metabolic rates.
Hopefully, others will understand why I asked permission for use of Randy Klabacka @HumbleHerper’s poster.
See for instance, this poster from my last presentation:
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.