Ribonucleotide incorporation enables repair of chromosome breaks by nonhomologous end joining
They invented a new term to obfuscate facts about food energy-dependent RNA-mediated DNA repair.
For comparison, see: A pinch of RNA spices up DNA repair
See also: First CRISPR babies: 6 questions that remain
CCR5 encodes a protein that some common strains of HIV use to infect immune cells. This gene interacts among microRNA-mediated networks of genes. That makes editing a threat to humanity if it promotes virulence in biophysically constrained HERVs.
See: CCR5 microRNA
See also: Ribosomal frameshifting in the CCR5 mRNA is regulated by miRNAs and the NMD pathway
Ask someone who knows how energy-dependent cell type differentiation occurs before editing genes that biophysically constrain viral latency. The facts about RNA-mediated cell type differentiation are too difficult for amateurs to understand.
James V. Kohl detailedl how quantized energy-dependent microRNA biogenesis links the pheromone-controlled physiology of reproduction to biophysically constrained viral latency and healthy longevity. Links from what organisms eat to the enzyme-dependent metabolism of food also link metabolic networks to microRNA-mediated genetic networks in the context of RNA interference. Drug therapies alter RNA interference by altering cell type differentiation in all cells of all individuals of all species. For comparison, during the past 26 years, Kohl has detailed how the chemistry of protein folding is biophysically constrained at every level of biological organization by conserved molecular mechanisms.
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